From 9787d73d444938667a5aab765d13b458c41ec35b Mon Sep 17 00:00:00 2001 From: lenoradickey32 Date: Sat, 20 Sep 2025 01:06:58 +0800 Subject: [PATCH] Add 'Glycogen Storage Disease (GSD)' --- Glycogen-Storage-Disease-%28GSD%29.md | 1 + 1 file changed, 1 insertion(+) create mode 100644 Glycogen-Storage-Disease-%28GSD%29.md diff --git a/Glycogen-Storage-Disease-%28GSD%29.md b/Glycogen-Storage-Disease-%28GSD%29.md new file mode 100644 index 0000000..43e1881 --- /dev/null +++ b/Glycogen-Storage-Disease-%28GSD%29.md @@ -0,0 +1 @@ +
Such a scheme serves as a template for the lactate shuttling current in the mammalian CNS. An almost contemporaneous report prolonged this scheme to apply to the mammalian mind, but with notable variations and additions. It was to prove some of the contentious papers in the field of brain vitality metabolism and the talk raging round the topic persists. The paper described experiments carried out utilizing cultures of mouse cerebral neurones and astrocytes, and demonstrated a few key features, which have been stoichiometrically strong (Pellerin and Magistretti, 1994). The background to the examine was the realization that if metabolic signaling were to occur between neurones and astrocytes in the mammalian CNS, there must exist a signaling mechanism whereby the neurones signal to astrocytes their want for supply of metabolic substrate. The signaling mechanism should have the next traits: (1) it should be associated to the frequency of exercise of the neurones i.e., the release of the compound must be associated to the activity of the neurones, (2) it should be sensed by the astrocyte i.e., there have to be mechanism(s) whereby the astrocyte senses the molecule, and (3) there should exist a system or course of whereby the focus of the molecule is decreased upon cessation of exercise i.e., there should be an efficient buffering system that reduces the focus of the molecule after exercise.

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